https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:31124 Wed 11 Apr 2018 16:45:05 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:14698 Wed 11 Apr 2018 16:15:41 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26497 Wed 11 Apr 2018 12:26:15 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:42445 1% predicted) and consumption of fried fish (OR -0.12, 95% CI -0.22, -0.01, P = 0.026) but not fish prepared by other cooking methods or estimated intakes of PUFA. There was no association between fish or PUFA intakes and COPD risk. Compared to age and sex matched controls, cases had poorer lung function and a higher rate of smoking prevalence but did not differ in their intakes of fish or PUFA or their PUFA levels in plasma phospholipids. In this sub-population, we found a marginally significant association between COPD risk and total long chain n-3PUFA levels in plasma phospholipids (OR 1.22 95% CI 1.00-1.49, P = 0.046). Given the relatively small number of cases in this analysis, this finding should be interpreted with caution, especially given the lack of association with other markers of n-3PUFA intake or status. Taken together, our data suggest that n-3PUFA intake and status are not determinants of improved lung function in this regional Australian population.]]> Tue 23 Aug 2022 10:54:28 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26603 Sat 24 Mar 2018 07:33:57 AEDT ]]>